When these two trials are considered together, it is uncertain how acetazolamide led to an improvement in physical signs of fluid retention in the ADVOR trial, since the drug produced only a modest diuretic effect in ADVOR and no natriuretic effect in DIURESIS-CHF
ADVOR: Acetazolamide in Acute Decompensated Heart Failure with Volume Overload
Was not significant if LVEF ≤40%, Female, GFR ≥40, >60mg lasix daily, more volume overload (congestion >4)
Excluded anyone on >80mg lasix, must be on 40 or more
New diagnosed HF were excluded from the trial so results cannot be applied to them
Congestion scoring system used in this study used variables which are reflective of extracellular dehydration but not intracellular dehydration
Further research can be directed to replicating efficacy demonstrated here, and preferably with multiple measurements like quantum of natriuresis, diuretic efficiency, 6 hour cumulative sodium excretion or the natriuretic response prediction equation (NRPE)
To select a population at high risk for loop diuretic resistance, at least one out of three additional inclusion criteria had to be fulfilled: (i) serum sodium ≤ 135 mmol/L; (ii) serum urea/Cr ratio > 50; or (iii) admission serum Cr increase of > 0.3 mg/dL compared to a previous measurement within 3 months.
The two groups did not differ with respect to the primary endpoint, that is, urinary sodium excretion in the 24 h following randomization, and they also did not differ with respect to the degree of clinical decongestion or changes in NT-proBNP after 72 h, but worsening renal function was more commonly seen in the acetazolamide group
There was no difference in two groups with respect to the risk of death or heart failure hospitalization within the next 90 days, although the investigators reported some divergence months after treatment had been stopped
STRONG-HF: Rapid up-titration of GDMT and close f/u reduced risk of 180-day all-cause death or HF readmission in AECHF admits